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2024-7-18 1:27:32
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A. C. Hurt et al.. Mutations I117V and I117M and Oseltamivir Sensitivity of Pandemic (H1N1) 2009 Viruses. Emerg Infect Dis 2011 Dec 19.  Full Text  
submitted by kickingbird at Dec, 20, 2011 from Emerg Infect Dis 2011 Dec 19 (via http://wwwnc.cdc.gov/eid/)
11-1079-ahead_of_print.pdf

Storms AD, Gubareva V, Su S, et al. Oseltamivir-resistant pandemic (H1N1) 2009 virus infections, United States, 2010-11. Emerg Infect Dis 2011 Dec 19.  Full Text  
submitted by kickingbird at Dec, 20, 2011 from Emerg Infect Dis 2011 Dec 19 (via http://wwwnc.cdc.gov/eid/)
11-1466-ahead_of_print.pdf

Hurt AC, Chotpitayasunondh T, Cox N, et al.. Antiviral resistance during the 2009 influenza A H1N1 pandemic: public health, laboratory, and clinical perspectives. Lancet Infect Dis 2011 Dec 19.  Abstract  
submitted by kickingbird at Dec, 20, 2011 from Lancet Infect Dis 2011 Dec 19 (via http://www.thelancet.com/journals/laninf/article/PIIS1473-30)
Influenza A H1N1 2009 virus caused the first pandemic in an era when neuraminidase inhibitor antiviral drugs were available in many countries. The experiences of detecting and responding to resistance ...

ROTH B, Mohr H, Enders M, Garten W, et al.. Isolation of influenza viruses in MDCK 33016PF cells and clearance of contaminating respiratory viruses. Vaccine. 2011.  Abstract  
submitted by kickingbird at Dec, 20, 2011 from Vaccine. 2011 (via http://www.ncbi.nlm.nih.gov/pubmed/22119922)
This paper summarizes results obtained by multiplex PCR screening of human clinical samples for respiratory viruses and corresponding data obtained after passaging of virus-positive samples in MDCK 33016PF ...

SAMMON CJ, McGrogan A, Snowball J, de Vries CS, et. Factors associated with uptake of seasonal and pandemic influenza vaccine among clinical risk groups in the UK: An analysis using the General Practice Research Database. Vaccine. 2011..  Abstract  
submitted by kickingbird at Dec, 20, 2011 from Vaccine. 2011. (via http://www.ncbi.nlm.nih.gov/pubmed/22133511)
Abstract BACKGROUND: Influenza vaccine uptake rates are low compared with uptake rates of many other vaccines. It is unclear how this differs between risk groups in the population and between pandemic ...

GILES BM, Bissel SJ, Craigo JK, Dealmeida DR, et a. Elicitation of anti-1918 influenza immunity early in life prevents morbidity and lower lung infection by 2009 pandemic H1N1 influenza in aged mice. J Virol. 2011.  Abstract  
submitted by kickingbird at Dec, 20, 2011 from J Virol. 2011 (via http://www.ncbi.nlm.nih.gov/pubmed/22130546)
The "Spanish" influenza pandemic of 1918 was responsible for 40 to 50 million deaths and is antigenically similar to the swine lineage 2009 pandemic influenza virus. Emergence of the 2009 pandemic from ...

KILLINGLEY B, Enstone JE, Greatorex J, Gilbert AS,. Use of a Human Influenza Challenge Model to Assess Person-to-Person Transmission: Proof-of-Concept Study. J Infect Dis. 2011..  Abstract  
submitted by kickingbird at Dec, 20, 2011 from J Infect Dis. 2011. (via http://www.ncbi.nlm.nih.gov/pubmed/22131338)
Background.?Influenza transmission in humans remains poorly understood. In particular, the relative contribution of contact, large droplet, and aerosol transmission is unknown. The aims of this proof-of-concept ...

SONG D, Moon H, An DJ, Jeong HY, et al.. A Novel Reassortant Canine H3N1 Influenza Virus between Pandemic H1N1 and Canine H3N2 Influenza Viruses in Korea. J Gen Virol. 2011.  Abstract  
submitted by kickingbird at Dec, 20, 2011 from J Gen Virol. 2011 (via http://www.ncbi.nlm.nih.gov/pubmed/22131311)
During recent canine influenza surveillance in South Korea, a novel canine influenza virus (CIV) H3N1 which was a putative reassortant between pandemic H1N1 2009 and H3N2 canine influenza viruses was isolated. ...

Nobuhiro Takemae, Sujira Parchariyanon, Ruttapong. Swine influenza virus infection in different age groups of pigs in farrow-to-finish farms in Thailand. Virology Journal 2011, 8:537.  Abstract  
submitted by kickingbird at Dec, 15, 2011 from Virology Journal 2011, 8:537 (via http://www.virologyj.com/content/8/1/537/abstract)
Background Understanding swine influenza virus (SIV) ecology has become more and more important from both the pig industry and public health points of views. However, the mechanism whereby SIV occurs ...

Epstein S. T-Cell Immune Responses and Asymptomatic H5N1 Influenza Infection. J Infect Dis 2011.  Abstract  
submitted by kickingbird at Dec, 3, 2011 from J Infect Dis 2011 (via http://jid.oxfordjournals.org/content/early/2011/11/10/infdi)
Approximately 60% of people ill with influenza A virus subtype H5N1 infection die [1], a case fatality rate about 25 times worse than that during the 1918 pandemic [2]. It is hard to even imagine the devastation ...

Powell TJ, Fox A, Peng Y, et al.. Identification of H5N1-Specific T-Cell Responses in a High-risk Cohort in Vietnam Indicates the Existence of Potential Asymptomatic Infections. J Infect Dis 2011.  Abstract  
submitted by kickingbird at Dec, 3, 2011 from J Infect Dis 2011 (via http://jid.oxfordjournals.org/content/early/2011/11/10/infdi)
Background.?Most reported human H5N1 viral infections have been severe and were detected after hospital admission. A case ascertainment bias may therefore exist, with mild cases or asymptomatic infections ...

Ma W, Lager KM, Li X, Janke BH, Mosier DA, Painter. Pathogenicity of swine influenza viruses possessing an avian or swine-origin PB2 polymerase gene evaluated in mouse and pig models. Virology. 2011 Feb 5;410(1):1-6. Epub 2010 Nov 11..  Abstract  
submitted by kickingbird at Nov, 28, 2011 from Virology. 2011 Feb 5;410(1):1-6. Epub 2010 Nov 11. (via http://www.ncbi.nlm.nih.gov/pubmed/21074235)
PB2 627K is a determinant of influenza host range and contributes to the pathogenicity of human-, avian-, and mouse-adapted influenza viruses in the mouse model. Here we used mouse and pig models to analyze ...

Song MS, Pascua PN, Lee JH, Baek YH, Park KJ, Kwon. Virulence and genetic compatibility of polymerase reassortant viruses derived from the pandemic (H1N1) 2009 influenza virus and circulating influenza A viruses.. J Virol. 2011 Jul;85(13):6275-86. Epub 2011 Apr 20.  Abstract  
submitted by kickingbird at Nov, 28, 2011 from J Virol. 2011 Jul;85(13):6275-86. Epub 2011 Apr 20 (via http://www.ncbi.nlm.nih.gov/pubmed/21507962)
Gene mutations and reassortment are key mechanisms by which influenza A virus acquires virulence factors. To evaluate the role of the viral polymerase replication machinery in producing virulent pandemic ...

Schrauwen EJ, Herfst S, Chutinimitkul S, Bestebroe. Possible increased pathogenicity of pandemic (H1N1) 2009 influenza virus upon reassortment. Emerg Infect Dis. 2011 Feb;17(2):200-8..  Abstract  
submitted by kickingbird at Nov, 28, 2011 from Emerg Infect Dis. 2011 Feb;17(2):200-8. (via http://www.ncbi.nlm.nih.gov/pubmed/21291589)
Since emergence of the pandemic (H1N1) 2009 virus in April 2009, three influenza A viruses-seasonal (H3N2), seasonal (H1N1), and pandemic (H1N1) 2009-have circulated in humans. Genetic reassortment between ...

US CDC. Limited Human-to-Human Transmission of Novel Influenza A (H3N2) Virus — Iowa, November 2011. MMWR 2011; 60 (dispatch); 1-3.  Abstract  
submitted by kickingbird at Nov, 25, 2011 from MMWR 2011; 60 (dispatch); 1-3 (via http://www.cdc.gov/mmwr/preview/mmwrhtml/mm60d1123a1.htm?s_c)
  DispatchNovember 23, 2011 / 60(Dispatch);1-3 On November 20, 2011, CDC confirmed three cases of swine-origin triple reassortant influenza A (H3N2) (S-OtrH3N2) virus infection in children in two ...

Calistri A, Salata C, Cosentino M, etc., al. Report of two cases of influenza virus A/H1N1v and B coinfection during the 2010/2011 epidemics in the Italian Veneto region. Virol J 2011 Nov 3;8(502).  Abstract  
submitted by kickingbird at Nov, 6, 2011 from Virol J 2011 Nov 3;8(502) (via http://www.virologyj.com/content/8/1/502/abstract)
From October 2010 to April 2011, in the Italian Veneto Region, 1403 hospitalized patients were tested for influenza virus infection by specific real time RT-PCR. Overall, 327 samples were positive for ...

Myers CA, Kasper MR, Yasuda CY, etc., al. Dual infection of novel influenza A/H3N2 in a cluster of Cambodian patients. Am J Trop Med Hyg 2011 Nov;85(5):961-3.  Abstract  
submitted by kickingbird at Nov, 6, 2011 from Am J Trop Med Hyg 2011 Nov;85(5):961-3 (via http://www.ajtmh.org/content/85/5/961.abstract?sid=8e64400d-)
During the early months of 2009, a novel influenza A/H1N1 virus (pH1N1) emerged in Mexico and quickly spread across the globe. In October 2009, a 23-year-old male residing in central Cambodia was diagnosed ...

Tsukamoto K, Javier PC, Shishido M, Noguchi D, Pea. SYBR Green Based Real-Time Reverse Transcription PCR for Typing and Subtyping of All Hemagglutinin and Neuraminidase genes of Avian Influenza Viruses and Comparison to Standard Serological Subtyping. J Clin Microbiol. 2011 Oct 26..  Abstract  
submitted by kickingbird at Oct, 30, 2011 from J Clin Microbiol. 2011 Oct 26. (via http://www.ncbi.nlm.nih.gov/pubmed/22031706)
Continuing outbreaks of H5N1 highly pathogenic (HP) avian influenza virus (AIV) infections of wild birds and poultry worldwide emphasize the need for global surveillance of wild birds. To support the future ...

Morimoto J, Sato K, Nakayama Y, Kimura C, Kajino K. Osteopontin Modulates the Generation of Memory CD8+ T Cells during Influenza Virus Infection. J Immunol. 2011 Oct 21.  Abstract  
submitted by kickingbird at Oct, 30, 2011 from J Immunol. 2011 Oct 21 (via http://www.ncbi.nlm.nih.gov/pubmed/22021613)
The adaptive immune system generates memory cells, which induce a rapid and robust immune response following secondary Ag encounter. Memory CD8(+) T cells are a critical component of protective immunity ...

Shinya K, Ito M, Makino A, Tanaka M, Miyake K, Eis. The TLR4-TRIF pathway protects against H5N1 influenza virus infection.. J Virol. 2011 Oct 26..  Abstract  
submitted by kickingbird at Oct, 30, 2011 from J Virol. 2011 Oct 26. (via http://www.ncbi.nlm.nih.gov/pubmed/22031950)
Pre-stimulation of the TLR4 pathway with lipopolysaccharide (LPS) protects mice from lethal infection with H5N1 influenza virus. Here, we reveal that the TLR4-TRIF pathway is required for this protective ...

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